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Critically ill patients are under consistent biological stress conditions, wherein lactate will be derived from anaerobic glucose metabolism (glycolysis), which produces energy 23 times faster than oxidative phosphorylation, and is vital to critical patients who are in an energy crisis [31]

The cAMP pathway in macrophages of ApoE / mice may partially explain this inhibition of chemokine and inflammatory factor secretion

A study showed that LPS administration caused a strong, dose- and time-dependent activation of NF-B across multiple organs, as demonstrated in transgenic NF-B reporter mice

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