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This is the process known to be responsible for significant damage to - and premature ageing of - our cells
Normal cells, which do not rely on FOXO4-p53 binding for survival, are unaffected
As mentioned previously, the TMAO is similar to that of oxidized low density lipoprotein (ox-LDL), which can promote the differentiation of monocytes into macrophages that scavenge ox-LDL and transform into foam cells (63)
Hepatology 2010;52(1):79-104
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