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141 This prolonged duration may be driven by albumin binding to the fatty acid residues of GLP-1 RAs and their ability to escape protease metabolism due to amino acid substitutions (Figure 1)
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Burgess S
Circ Res 134(4):425441 Wang H, Cai P, Yu X, Li S, Zhu W, Liu Y, Wang D (2024) Bioinformatics identifies key genes and potential drugs for energy metabolism disorders in heart failure with dilated cardiomyopathy
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