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Several peptides being studied have mechanisms that directly address this core problem: they promote new blood vessel formation (angiogenesis), reduce the inflammatory cascade that accelerates disc breakdown, and stimulate the cellular repair processes needed to rebuild damaged disc structures

HUBERMAN: It is clear that BPC-157 can exit the gut, but how it gets traffic to particular sites within the body that are injured still isn't clear

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Historical exposure to stress may therefore alter cellular responses to aripiprazole toxicity