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Pathophysiologic Context In PAH + HFpEF overlap: * ETA receptors drive vasoconstriction, hypertrophy, and fibrosis * ETB receptors mediate endothelin clearance, nitric oxide release, and natriuresis * HFpEF patients rely on ETB signaling to maintain volume balance and endothelial function Blocking ETB in HFpEF increases the risk of fluid retention and rising leftsided filling pressures
Results in OASIS 1 were broadly similar to those seen with injectable semaglutide 2.4 mg in the STEP series of trials
Similarly, the degree of lipid peroxidation evaluated by BODIPY staining was consistent with the change in the total ROS level
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