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A 2015 review in Biomedical Research International confirmed GHK-Cus role in accelerating wound healing, stimulating collagen synthesis, and modulating gene expression related to tissue repair

It is called glucotoxicity previously, 126 however, we may call this phenomenon hyperglucotoxicity, as a normal glucose level is not toxic to islet cells

(5) These increases were associated with a marked decrease in oxidative stress and improvements in immune function, as indicated by a better oxidized (GSSG) to reduced (GSH) glutathione ratio and increased natural killer cell activity