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GSH depletion can trigger ferroptosis, which has been exploited in cancer treatments [17], and supplementation with GSH is utilized by glutathione peroxidase 4 (GPX4) to prevent lipid peroxidation, in turn inhibiting ferroptosis

Its potential to modify underlying disease pathology, rather than just block a final inflammatory product, merits further investigation in clinical settings

This protection allows a higher percentage of intact glutathione to reach the small intestine for absorption