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Fras JP, et al
[2] suggested that BPC-157 may assist repair by supporting tendon fibroblast migration and spreading in laboratory studies, with researchers observing that "F-actin formation as detected by FITC-phalloidin staining was induced in BPC 157-exposed cells," [2] with activation of focal adhesion signaling through phosphorylation of FAK and paxillin proposed to assist cell attachment and movement within the extracellular matrix in these settings
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