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CAF22 e NfL sono biomarcatori associativi: non costituiscono una dimostrazione anatomica di degenerazione della giunzione neuromuscolare causata dal farmaco

Considering the broader genetic background, pharmacogenomic variability may further contribute to treatment heterogeneity, as genetic variants affecting GLP-1 receptor signaling, appetite regulation, insulin secretion, and hostmicrobiota interactions could influence individual responsiveness (126)

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BACE1 physiological functions may limit its use as therapeutic target for alzheimers disease