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Sharing a 94% structural homology with native human GLP-1, Semaglutide is distinguished by three modifications: 1) amino-acid substitutions at position 8 (alanine to -aminoisobutyric acid or Aib) and position 34 (lysine to arginine), and acylation of the lysine in position 26 with a spacer consisting of two 8-amino-3,6-dioxaoctanoic acid (ADO) moieties, a glutamic acid moiety, and a C-18 fatty di-acid side chain (Lau et al., 2015)
The synergy of GIPR and GLP-1R signaling within the pancreas, adipose tissue, central nervous system, and potentially the liver provides a robust framework for how tirzepatide could help reduce the progression or outcome of MASLD
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Benefits include: \Angiogenesis: May improve blood vessel formation and circulation (Animal Study)
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