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Therefore, AdoMet supplementation may not alter GSH status in rodents who already have innate compensatory mechanisms for overcoming methionine dysfunction [86]
In another study, IGFBP-3 was able to enhance etoposide-induced cell growth inhibition by blocking the NF-B signaling pathway in gastric cancer cells, confirming that IGFBP-3 has become a key target and marker for cancer therapy and further exploration of its in-depth regulatory mechanisms is worthwhile (91)
A., Yamazaki, S., Tei, H., Sakaki, Y
Both the megadose amount and the long term maintenance dose (up to 20g/day) seems unnecessarily high to me
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