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Separating these readouts in future trials will be essential to determine whether GLP agonism produces true reparative effects versus indirect metabolic benefits
Ltd) and retatrutide are the most advanced and are in Phase III clinical trials (Table 2)
One possible explanation for the similar effects on weight loss observed with both GIPR agonism and antagonism might be that chronic pharmacological GIPR activation induces functional antagonism in a subset of neurons through receptor desensitization, as seen in adipocytes, isolated pancreatic islets and mouse neuroblastoma Neuro2a cells 36,37
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