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These fascinating results form a basis for a targeted intervening in the metabolism of immune cells and for developing a new generation of immunotherapies, added Markus Ollert, Ph.D., a professor at and director of the Department of Infection and Immunity at LIH

GHK-Cu does not directly interact with cytoskeletal proteins but instead operates through copper-dependent enzymatic activation and transcriptomic reprogramming

This metabolic transformation also results in reduced affinity of the compound for its cellular target