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Nicotinamide adenine dinucleotide (NADH) and acetyl-CoA, two metabolic intermediates produced in the course of FAO, induce activation of PDK, which in turn phospho-inactivates the E1 subunit of the PDH complex, leading to lower rates of glucose oxidation and higher rates of lactate release 70

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Our data, from a reasonably large sample size, validates the significance of GSH deficits in SZ pathology, which has been suggested previously

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