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Under conditions of hypoxia, the low levels of oxygen lead to accumulation of NADH and under these conditions mitochondria can increase the oxidation of NADH via the LDH-mediated reduction of pyruvate
Theoretical and mechanistically predicted concerns for example, the consequences of chronically stimulating the GH/IGF-1 axis, or the metabolic implications of ghrelin-pathway activation and appetite stimulation have not been resolved by long-term human trials
It's not cheap, but the dosage and clinically backed ingredients for appetite support, healthy blood sugar, metabolic and heart health support justify the cost
Santocchi et al., 2020
