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The oxidized glutathione (G-S-S-G) produced is reduced by glutathione reductase, using NADPH as the source of electrons

Pain 153 , 24482457 (2012)

Oxidative stress also activates the Kelch-like ECH-associated protein 1 (Keap1)-Nuclear factor erythroid 2-related factor 2 (Nrf2)-antioxidant response element (ARE) redox regulator pathway, releasing Nrf2 to regulate the expression of genes that control antioxidant, inflammatory and immune system responses, facilitating GSH activity

The pathophysiology of AD is complex, combining genetic predisposition and impaired epidermal barrier function with an unbalanced inflammatory response to environmental factors (1, 18)