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Reported cellular endpoints include endothelial and keratinocyte migration, collagen deposition, integrin-linked kinase and Akt activation, and HIF-1 alpha interaction in migration assays
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The most important finding in our study is that TXR treatment has a neuroprotective effect in the KA-induced excitotoxicity model by providing significant improvements in cerebral cortex MDA and GSH levels, decreasing cerebral cortex IL- 1 levels and decreasing Gal- 3 expression in the CA1 region of the hippocampus and various layers of the cerebellum
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