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To address this question, we mutated Tyr110 in the CPYS-motif of ScGrx7 as a candidate residue that might contribute to the glutathione activator site (see Discussion for details) and performed a preliminary study with wild-type ScGrx7 as a control
Millar et al, 1999) in association with aberrant DNA methylation of its CpG island associated promoter and first exon (Millar et al, 1999)
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