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By inhibiting JAK1, JAK2, JAK3, and TYK2, delgocitinib modulates a wide array of cytokine pathways integral to CHEs diverse immunopathogenesis, including Th1, Th2, Th17, and Th22 -mediated inflammation [102, 103]
Experimental vascular studies indicate that BPC-157 modulates endothelial nitric oxide synthase activity by altering the interaction between endothelial nitric oxide synthase and its regulatory binding partner caveolin-1 [1][2]
Cell Metab 15:675690
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