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Many individuals with type 1 diabetes gain weight due to intensive insulin therapy, so adding a GLP-1 agent may help counter this effect
Glucagon enhances multiple steps in this pathway: Increased lipolysis: Glucagon activates hormone-sensitive lipase in adipose tissue, releasing more fatty acids into circulation Reduced lipogenesis: Glucagon suppresses the enzymes responsible for creating new fat from carbohydrates Enhanced hepatic oxidation: Glucagon upregulates liver enzymes involved in fatty acid oxidation Ketogenesis: When fat oxidation exceeds the citric acid cycle capacity, excess acetyl-CoA is converted to ketone bodies, an alternative fuel source The net effect is a metabolic state favoring fat breakdown over fat storage
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Various studies have looked at DSIP and found somewhat inconsistent results (Pollard BJ, 2001)
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