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Evidence supports the role of dysbiosis in MS, particularly on immune regulatory pathways
The mechanisms likely involve both the hormonal disruption of rapid fat loss (fat tissue helps regulate oestrogen and testosterone) and the direct effects of GLP-1 receptor activation on dopamine-mediated sexual motivation
Furthermore, membrane phospholipids bound to polyunsaturated fatty acids (PUFAs) are highly sensitive to free radical damage, leading to the accumulation of lipid peroxides (such as LOOH) [52]
Supplementary Data 2 (download ZIP Original pictures of cFOS and Cy5 drug appearance shown in Extended Data Fig
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