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glutathione reductase (GR) then regenerates GSH from GSSG using NADPH
They likely reflect the difference between pathological IGF-1 excess (which does appear to increase cancer risk and accelerate ageing in some models) and normalisation of IGF-1 in individuals whose levels have declined below an optimal physiological range due to somatopause
The CRM 13180 PDX model was provided by the laboratory of Dr Judith Leopold (University of Michigan) 61
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