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Exosome-transmitted lncARSR promotes sunitinib resistance in renal cancer by acting as a competing endogenous RNA
Reported activities include fibroblast stimulation, controlled intracellular copper delivery, modulation of collagen and glycosaminoglycan synthesis, and broad changes in gene expression across stress-response and tissue-repair networks
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The additional cost and complexity seem justified for chronic cases, though straightforward acute injuries may respond perfectly well to BPC-157 alone
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