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Post activation of cellular senescence, senescent cells release a variety of cytokines, chemokines, and matrix metalloproteinases, which causes the emergence of senescence-associated secretory phenotype (SASP) (Beck et al

The extraordinarily high levels of GSSG in these plants could indicate the pivotal role of glutathione in restoring TMV resistance in the background of SA deficiency, including suppression of oxidative stress (HR) in virus-infected tissues and regulating additional downstream defense responses

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