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Its mechanism involves activating the AMPK/SIRT1 pathways and suppressing lipogenic regulators like PPAR and SREBP-1c, highlighting its potential as a therapeutic candidate for NAFLD (96)
A significant number of people unintentionally develop liver damage from acetaminophen, even at doses just exceeding the recommended daily limit of 4 g
5, when PC3 cell lines were treated with SBA-15 MSN and FA-SNCD@SBA-15 at concentrations up to 50 g mL 1 for a duration of 24 h, the cell viability remained above 90%
ACTH-related pathways)
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