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Keap1 reactive cysteine residues have a negative impact on Keap1-mediated Nrf2 enzymatic activity, which results in Nrf2 accumulation/activation and cytoprotection by enhanced ARE transcriptional genes (Dinkova-Kostova et al., 2005a)
Recently, an interesting study revealed that fructose at a high level triggered progressive glomerular injury by initiating ferroptosis and reported that mitochondrial single-strand DNA-binding protein 1 (SSBP1) is an important regulator in the glomeruli of high fructose-fed rats, as determined via quantitative proteomic analysis
This suggests T4 might play a role in disease pathogenesis or tissue repair
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