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Adding cagrilintide to retatrutide creates a four-receptor approach (GLP-1, GIP, glucagon, and amylin)
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This metabolic dysbiosis can cause the imbalance of antioxidant defense systems and apparently raising the risk of periodontal tissue destruction (Shang et al., 2023)
Behind this phenomenon is the deep intervention of GLP-1 in the "central plan" of cellular energy metabolism: it activates GLP-1 receptors, reprograms cellular energy distribution pathways, and shifts the body from an "energy wasting" metabolic mode to an "efficient and economical" one, thereby achieving the dual goals of disease treatment and health improvement
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