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In another study using rabbit disc explants, a traumatic disc event without structural damage to the NP, AF and endplate caused transcriptional up-regulation of pro-inflammatory cytokines and catabolic enzymes
Here is how it works: It works by mimicking natural satiety signals, making it easier to eat less, feel full, and stay consistent
It binds to a particular family of receptors called melanocortin receptors and helps regulate a number of bodily functions [2]
But healing is not just about making more collagen
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