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While the targeted residue(s) is(are) unknown, AtAPK1 and AtAPK4 are likely subject to persulfidation ( in vitro in the presence of GSSG, suggesting protein glutathionylation ( C
Instead, low glutathione in the rest of the body could be due to an increased use of it for combating oxidative stress.[ref] Neurodegenerative diseases: Alzheimers, Parkinsons, and MS are all characterized by neuronal injury from oxidative stress
Wiesweg, B., Johnson, K
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